Contact allergy to cinnamon case report Assignment

Contact allergic reaction to cinnamon case report - Assignment Examplenickel, palladium, gold and mercury used in dental amalgam may also cause contact stomatitis.Allergic contact stomatitis is classified as sheath IV hypersensitivity reaction and prone to people who have previously been sensitized to the allergen. Its reaction falls beneath delayed hypersensitivity reaction as it several hours or even a day for the antigen reaction to call on evident. The allergic process occurs in two phases namely the founding phase and the effecter phase. In the induction phase, the allergens which have infiltrated and bound themselves to epithelial protein thus forming complexes are phagocytised by macrophages, specialized cells. The cells then move to regional ganglia stimulating production of the memory T lymphocytes. These lymphocytes are responsible for keeping memory of the antigen so as to trigger a more rapid immune response in the event of subsequent occurrences of the antigen. The effector phase is characterised with the activation and production of T lymphocytes (CD4 cells) by the cytotoxic T lymphocytes (CD8 cells) produces in the first phase. The cytotoxic T lymphocytes bind to the epithelial cells causing the death of cells that present the complexes. There are two introductory reasons as to why this condition is not common despite high rates of exposure to the allergens first, spitting ensures constant cleaning of the mucous membrane thus minimizing contact time with the allergen and, second, the high degree of vascularisation of the mucosa increases the rate of antigen absorption, reducing further prolonged exposure to these substances.Contact stomatitis manifest in a government issue of symptoms which include tissular edema, erythema, cracking, ulcerative areas, hyperkeratosis, desquamation, and vesicles. They manifest in the area that is exposed to the causal agent and may be with or without pain. Its appearance is determined